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我試圖在文檔中突出顯示特定「細胞因子」一詞的術語。在文檔中有幾個這個詞的例子。我試着這樣做：Solr - 突出顯示
http://localhost:8983/solr/Cytokine/select?q=cytokine&hl=true&hl.fl=content
在我得到的結果是：
<response>
<lst name="responseHeader">
<int name="status">0</int>
<int name="QTime">1</int>
<lst name="params">
<str name="q">cytokine</str>
<str name="hl">true</str>
<str name="hl.fl">content</str>
</lst>
</lst>
<result name="response" numFound="1" start="0">
<doc>
<arr name="title">
<str>
High Glucose Increases the Expression of Inflammatory Cytokine Genes in Macrophages Through H3K9 Methyltransferase Mechanism
</str>
</arr>
<arr name="content">
<str>
Recent studies ... SNIP ...in macrophages.
</str>
</arr>
<str name="id">c656ee36-bab5-4204-975d-80c720b50ec0</str>
<long name="_version_">1517203104151371776</long>
</doc>
</result>
<lst name="highlighting">
<lst name="c656ee36-bab5-4204-975d-80c720b50ec0"/>
</lst>
</response>
下面是文檔：
{"title":"High Glucose Increases the Expression of Inflammatory Cytokine Genes in
Macrophages Through H3K9 Methyltransferase Mechanism","content":"Recent studies suggest that histone modification is one of the mechanisms
regulating inflammatory cytokine gene expression in hyperglycemic conditions.
However, it remains unknown how histone methylation is initiated and involved in
changes of inflammatory cytokine gene expression under high glucose (HG)
conditions. Our aim was to investigate whether H3K9 methylation was involved in
HG-induced expression of inflammatory cytokines in macrophages. Expression
profile of cytokine genes under hyperglycemia in THP-1-derived macrophages was
determined by human cytokine antibody array. Based on the results from the human
cytokine antibody array analyses, the H3K9me3 levels of 4 inflammatory cytokine
genes, including interleukin-6 (IL-6), IL-12p40, macrophage inflammatory
protein-1α (MIP-1α), and MIP-1β under HG were determined by ChIP assays.
Furthermore, the expression of these 4 inflammatory cytokine genes under either
HG or chaetocin (an inhibitor of SUV39H1 methyltransferase) exposure or
overexpression of SUV39H1 (a H3K9me3-specific methyltransferase) was analyzed by
quantitative polymerase chain reaction. Macrophages cultured in HG conditions
showed increased gene expression and decreased H3K9me3 levels of inflammatory
cytokine genes compared with macrophages incubated in normal glucose (NG)
culture. Inhibition of SUV39H1 with chaetocin in NG-treated macrophages also
increased the expression of IL-6, IL-12p40, MIP-1α, and MIP-1β. Furthermore,
inhibition of SUV39H1 with chaetocin in HG-treated macrophages further increased
the expression of these inflammatory cytokines. Contrarily, NG-treated
macrophages transfected with SUV39H1 plasmids show decreased expression of
inflammatory cytokines. Furthermore, overexpression of SUV39H1 in HG-treated
macrophages alleviated the expression of inflammatory cytokines under HG
conditions. Finally, HG also increases the expression of inflammation cytokines
in mouse bone marrow-derived macrophages. Our data demonstrated that HG increases
the expression of inflammatory cytokines in macrophages through decreased H3K9me3
levels, which was partly mediated by SUV39H1. Dysregulation of epigenetic histone
modification may be one of the underlying mechanisms for HG-induced inflammatory
cytokine expression in macrophages."}
我在做什麼錯？